RT @NeuroscienceSAS: RPS25 cam be a potential therapeutic target for C9orf72-related amyotrophic lateral sclerosis. #neuroscience #brain #n…
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
@pjobphd For RPS25, at least one recently-reported really interesting phenotype was the attenuation of C9orf72 dipeptide repeat production: https://t.co/SGtRapvpFr
RPS25 cam be a potential therapeutic target for C9orf72-related amyotrophic lateral sclerosis. #neuroscience #brain #neuron #amyotrophiclateralsclerosis #ALS #neurodegenerativediseases https://t.co/Mt5G7WAp5f
RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeats | Nature Neuroscience https://t.co/SEkFzIM86e
RT @realToddLab: New article about RAN translation modifiers from Gitler lab. Worth a read! https://t.co/n7jBDhzd6H
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
#RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeats https://t.co/I5FWA6BqSh #ALS
RT @realToddLab: New article about RAN translation modifiers from Gitler lab. Worth a read! https://t.co/n7jBDhzd6H
RT @realToddLab: New article about RAN translation modifiers from Gitler lab. Worth a read! https://t.co/n7jBDhzd6H
RT @realToddLab: New article about RAN translation modifiers from Gitler lab. Worth a read! https://t.co/n7jBDhzd6H
RT @realToddLab: New article about RAN translation modifiers from Gitler lab. Worth a read! https://t.co/n7jBDhzd6H
New article about RAN translation modifiers from Gitler lab. Worth a read! https://t.co/n7jBDhzd6H
RT @NeuropathClinic: RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide r…
Trending on PubMed: RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeats. https://t.co/e2sK8nYcw1
RT @Montibeller_GG: RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide re…
RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeats. #neuropath https://t.co/C4l7uUj8ux
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
RT @MoensT: Loss of RPS25 suppresses RAN translation! https://t.co/Au1dVEXDX9
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
RT @ShorterLab: RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeat…
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
Cool!
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
RT @Montibeller_GG: RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide re…
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeats https://t.co/3A8lCet9Ro
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
@NonselfMutation
RT @NatureNeuro: The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins…
The most common cause of #ALS and #FTD is C9orf72 expansion that causes unconventional (RAN) translation of toxic proteins; a screen for regulators of RAN translation identified the ribosomal protein RPS25, suggesting a therapeutic target https://t.co/kn2l
@StanfordMed demonstrating how #iPSCs can contribute to the search for new drug targets. In a paper published in @NatureNeuro, the Gitler Lab have managed to identify a key gene (RPS25) behind the clumps of toxic proteins found in ALS. https://t.co/4wq26b0
RT @DrDiegoClemente: Great new data about ALS development. @NatureNeuro https://t.co/wbO01p9lEr
RT @DrDiegoClemente: Great new data about ALS development. @NatureNeuro https://t.co/wbO01p9lEr
Great new data about ALS development. @NatureNeuro https://t.co/wbO01p9lEr
Could turning off, or blocking, RPS25 gene cure #ALS (Lou Gehrig's disease) and other neurodegenerative illnesses (#huntingtonsdisease and spinocerebellar ataxia)? https://t.co/PKG816o6dL Looks like an interesting avenue to pursue.
RT @dutassoni: New paper from the Gitler Lab! Congrats to all the authors, especially Shizuka for the excellent work! #ALS #RANtranslation…
RT @ShorterLab: RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeat…
Great new paper by Aaron Gitler! RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeats @StanfordBrain https://t.co/hBmGWpH4Lf
Scientists at @StanfordMed performed a genetic screen for regulators of RAN translation and identified RPS25, presenting a potential therapeutic target for C9orf72-related #ALS and frontotemporal #dementia https://t.co/ux88NXVSKG
RT @ShorterLab: RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeat…
RT @dutassoni: New paper from the Gitler Lab! Congrats to all the authors, especially Shizuka for the excellent work! #ALS #RANtranslation…
RT @ShorterLab: RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeat…
New paper from the Gitler Lab! Congrats to all the authors, especially Shizuka for the excellent work! #ALS #RANtranslation #C9orf72 https://t.co/I5vtAgLd6V
RT @ShorterLab: RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeat…
RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeats https://t.co/2vv7dFNrf4
RT @ShorterLab: RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeat…
RPS25 is required for efficient RAN translation of C9orf72 and other neurodegenerative disease-associated nucleotide repeats, great work from ADG, LP, @isaacs_adrian et al: https://t.co/yfh99SQ6XM
RPS25: a new potential therapeutic target for #C9orf72 #ALS and #FTD? https://t.co/SfVV9tLsKP #GitlerLab