RT @TristanRutland7: 16/ However in things like stem cells, we want these cells to keep dividing forever (as they give rise to all our cell…
16/ However in things like stem cells, we want these cells to keep dividing forever (as they give rise to all our cells). 17/ When cells become neoplastic, having TERT active allows them to divide forever, which is obviously bad! https://t.co/ZRHw5zsK38
@Tinfoilhatter @DarkPilled @sailorrooscout telemorase normally is what creates the recurring stop codon on the 3' section of an RNA strand. Tumors use hTERT to effectively create shortcuts through the checkpoints found in mitosis, allowing cancer cells t
New in Pubmed: "Mechanisms of human telomerase reverse transcriptase (hTERT) regulation: clinical impacts in cancer." https://t.co/buESXO9iGc
Mechanisms of human telomerase reverse transcriptase (hTERT) regulation: clinical impacts in cancer. https://t.co/Gz8xqdWILz
Mechanisms of human telomerase reverse transcriptase (hTERT) regulation: clinical impacts in cancer. https://t.co/zDDYSkNOEm
New in Pubmed: "Mechanisms of human telomerase reverse transcriptase (hTERT) regulation: clinical impacts in cancer." https://t.co/0DqOnlE8n6
RT @RicardoLeao_MD: Mechanisms of human telomerase reverse transcriptase (hTERT) regulation: clinical impacts in cancer https://t.co/EHWqTh…